Due to the prominent role of IFNγ in enhancing phagocyte antimicrobial effector functions [14], we first blocked IFNγ actions in iKIR-treated and infected mice using an anti-interferon gamma receptor (IFNGR) antibody or IgG control antibody before infection and treatment with either iKIR or scrambled KIR peptide. This evidence concerns the gene IFNG and infection.