Our data show that iKIR enhances specifically the production of pro-inflammatory cytokines known to drive proper abscess formation and improved infection outcome (IL-1β) [3], neutrophil recruitment (CXCL1 and CXCL2) [51], but not monocyte recruitment (CCL2) [52] in response to MRSA skin infection (Figs 3B and S2A). The gene discussed is CXCL2; the disease is skin infection.