CXCL10 and type 1 diabetes mellitus: GABA inhibited human PBMC proliferative responses and nuclear factor (NF)-κB activation13,18, as well as secretion of IL-6, TNF, IL-17A, CXCL10/IP-10, CCL4, CCL20, and MCP-3 from anti-CD3 stimulated PBMC from T1D patients18.