Interestingly, only the later, second peaks were greater in CFTRtm1HGU animals when compared to their wild‐type controls with pancreatitis, suggesting the direct response to supramaximal CCK stimulation of acinar cells did not differ from controls in the pancreas of CFTR‐disrupted animals (Figure 2A‐C). The gene discussed is CFTR; the disease is pancreatitis.