Bacteria can break the alveolar wall (causing leakage of protein fluid into the alveoli), block pulmonary blood vessels, activate platelet-activating factor receptors, change the permeability of endothelial cells, and mediate the activity of Src family kinases, thereby making the lungs susceptible to parasitic infections and forming parasitic pneumonia (Cheepsattayakorn and Cheepsattayakorn, 2014). This evidence concerns the gene SRC and parasitic infectious disease.