To this regard, a previous study in a murine model of BPD suggested a possible set of differentially expressed proteins in BPD (i.e., p-AMPKb1[S108], platelet-derived growth factor receptor PDGFRb, and SLUG) based on their relevance in developmental lung injury and such set was further validated on a cross-platform transcorrelation analysis (protein/transcript) (56). This evidence concerns the gene PDGFRB and bronchopulmonary dysplasia.