To further characterize how the loss of distinct α2δ subunits affects postnatal development and neurological disease we next assessed body and brain weights of juvenile (3-4-week-old; α2δ-2/-3 double knockout and ducky) or adult (8–13-week-old; α2δ-1/-3 double knockout and ducky) male mice. The gene discussed is ATXN2L; the disease is nervous system disorder.