SOAT1 and COVID-19: In the advanced stage of the disease, critically ill COVID‐19 patients develop a cytokine storm where many inflammatory mediators using the JAK/STAT signaling pathway such as IL‐6, IFN‐γ, the granulocyte colony‐stimulating factor (G‐CSF) or IL‐2, and chemokines result in an influx of macrophages and neutrophils damaging the lung tissue.