However in our case, the role of hepatitis C in rhabdomyolysis remains unclear since muscle biopsy showed no evidence for myositis, there was only a very distant history of illicit drug abuse during his teens, but not for decades [based on the history and repetitive urine analysis tests over the years] and he never received therapy for hepatitis C. It is conceivable that hepatitis C in combination with genetic disorder with predisposition to rhabdomyolysis, such as SCA2, was the cause for these repetitive attacks of rhabdomyolysis. Here, ATXN2 is linked to hereditary disease.