Studies have shown that intracranial germ cell tumors may develop through two different pathogenesis: one is KIT/RAS changes, increased KIT mRNA expression and severe chromosomal instability, and the other is the absence of any of the above abnormal unknown mechanisms, while the presence of KIT/RAS changes is usually associated with the upregulation of KIT mRNA and chromosomal instability. Here, KIT is linked to testicular germ cell tumor.