Our results are consistent with the FKBP5 gene-environment interaction model of schizophrenia, according to which childhood adversity leads to reduced DNA methylation at GREs of FKBP5, which results in up-regulation of FKBP5 and down-regulation of NR3C1, as we detect in the cerebellum of the patients. This evidence concerns the gene FKBP5 and schizophrenia.