This was done via activation of Wnt/β-catenin pathway and downregulation of GSK3β, causing increased miR-135a, β-catenin, cyclinD1, vimentin mRNA, and protein expression, whereas decreased GSk3β, E-cadherin mRNA, and protein expression in BLCA tissues compared to the adjacent normal tissue [37]. The gene discussed is GSK3B; the disease is bladder transitional cell carcinoma.