Female patients with PWS showed analogous patterns with low levels of inhibin B and anti-Mullerian hormone (AMH) (both produced by the granulosa cells in follicles during primary and preantral stages) with normal FSH levels and normal size of primordial and antral follicle pool; only a small percentage of patients showed normal or slightly decreased inhibin B and AMH levels with the development of a dominant follicle [45,46,47]. Here, AMH is linked to Prader-Willi syndrome.