Interestingly, we did not observe any significant changes in microtubule gliding velocities, neither in the presence of recombinant wildtype FUS-GFP or FUS-P525L-GFP nor in the presence of whole‐cell lysates obtained from ALS-patient-specific iPSC-derived spinal motor neurons which endogenously express these FUS variants. Here, FUS is linked to amyotrophic lateral sclerosis.