They found lower FGF-23, and higher Klotho in the LPD + KA group compared to the LPD one, concluding that KA supported the nutritional status and was able to correct FGF-23 and Klotho abnormalities involved in cardiovascular calcification and cardiac remodeling decrease in CKD [24]. The gene discussed is KL; the disease is disseminated peritoneal leiomyomatosis.