Furthermore, another study reported that both full-length and truncated HMGA2 induce the development of various benign tumors in mesenchymal tissues such as breast fibroadenoma, salivary gland adenoma, and preputial gland hyperplasia, suggesting that misexpression of HMGA2 itself is sufficient for tumorigenicity in this cell linage [24]. The gene discussed is HMGA2; the disease is benign neoplasm.