More recently, neutrophil extracellular traps (NETs), a novel cell death mechanism initially described to fight infectious agents by releasing web‐like structures composed of DNA, specifically modified citrullinated histones and cytoplasmic degradative enzymes (including elastase, matrix metalloprotease, and myeloperoxidase), have recently been shown to play a role in promoting tumour progression and tumour‐associated thrombosis in various cancers [17, 18, 19, 20, 21]. The gene discussed is MPO; the disease is neoplasm.