Firstly, data on virus antibodies and proinflammatory cytokines (e.g., interleukin [IL]-1, IL-6, IL-8 and tumor necrosis factor-α) were not available, which would provide a proper insight into the pathophysiological stage of the myocardial injury from viral infection to the immune reaction. The gene discussed is CXCL8; the disease is viral infectious disease.