Both the male and female myocardial injury patients had higher levels of creatine kinase-MB, lactic dehydrogenase, α-hydroxybutyrate dehydrogenase, hs-cTnI, brain natriuretic peptide (BNP), myoglobin, leucocytes, high-sensitive C-reactive protein (hs-CRP), urea nitrogen, aspartate aminotransferase, prothrombin time and D-dimer but a lower lymphocyte percentage and monocyte percentage (all p < 0.05) (Supplementary Table S1). The gene discussed is TNNI3; the disease is injury.