We then generated BiPSCs carrying the most frequent reciprocal cTr t(11;14) in MM (BiPSC13 t(11;14) and MIB2-6 t(11;14)), and also generated BiPSC13 t(11;14) with p53 deletion, which is involved in the progression of MM. Here, MIB2 is linked to Miyoshi myopathy.