Another study in neuroblastoma, the authors showed similar phenomenon and suggested that this function of B4GALT3 is driven by glycosylation of integrin beta 1 resulting in extended retention of matured beta 1 integrin and increased phosphorylation of focal adhesion kinases (p-FAK) consequently leading to enhanced tumor cell migration and invasion [36]. The gene discussed is B4GALT3; the disease is neuroblastoma.