VEGF‐expressing MSCs have been reported to promote angiogenesis and improve the infarct size after a month by 10% in rats98 and 30% in sheep99 post‐MI, whilst GATA‐4 triggered antiapoptotic pathways when overexpressed in MSCs, displaying a threefold increase in ejection fraction and fractional shortening compared to unmodified MSCs in mice after MI.100. Here, GATA4 is linked to myocardial infarction.