Given that the dysregulation of the JAK/STAT signaling pathway plays an instrumental role in breast cancer development and metastasis as well as in other types of cancer [23, 24, 25, 26, 27, 28, 29], we concluded that C/EBPβ promotes TNBC carcinogenesis probably via the regulation of the JAK/STAT signaling pathway. This evidence concerns the gene SOAT1 and breast cancer.