As shown in Fig. 7a, the 10 KEGG pathways that were positively correlated to the high expression of TUBA1C were as follows: cell cycle, p53 signaling pathway, basal transcription factors, ubiquitin mediated proteolysis, glycolysis gluconeogenesis, citrate cycle TCA cycle, oxidative phosphorylation, pancreatic cancer, renal cell carcinoma, bladder cancer. Here, TUBA1C is linked to pancreatic neoplasm.