Most common pathogenic variants in DCM patients are identified in genes encoding sarcomere proteins (e.g., Titin (TTN) and Myosin Heavy Chain 7 (MYH7)); Z-disk components (e.g., Filamin-C (FLNC) and BLC2 Associated Athanogene 3 (BAG3)); and in the LMNA gene, encoding a structural protein of the nuclear envelope [10,11,12]. This evidence concerns the gene TTN and familial dilated cardiomyopathy.