Furthermore, we found that the fatty acid translocase (FAT)/CD36 (Figures 4G, 4H, 5A), and genes involved in fatty acid synthesis (FAS) (Figure 5B) and fatty acid β-oxidation (proliferator-activated nuclear receptor, PPAR-α) (Figure 5C–5E) were significantly upregulated in the liver of NAFLD mice, whereas they were lowered after swimming exercise. This evidence concerns the gene CD36 and metabolic dysfunction-associated steatotic liver disease.