GSN and hereditary amyloidosis: Blood samples were collected from the proband and family members to obtain DNA.Genetic analysis in the proband included polymerase chain reaction (PCR)amplification and Sanger sequencing of the most frequently mutated exons 4, 11, 12,13 and 14 of TGFBI as well as the gelsolin (GSN)pathogenic variant c.640G>A, which in Finland causes familial amyloidosis,Finnish type (FAF) with corneal lattice amyloidosis,5 using standard HUSLAB protocols.