In addition to CXCL12 overexpression, senescent tumor cells can inhibit the infiltration of CD8+ T cells into tumor tissues through insufficient production of T cell‐attracting chemokines, physical barriers, or aberrant vasculature.[42, 43, 44] CXCL9, CXCL10, and CXCL11 have a major role in attracting tumor‐specific T cells into cancers,[45, 46] and the insufficient expression of these chemokines at the tumor favors immune evasion.[47] In this study, the levels of CXCL9 and CXCL11 were insufficient, and CXCL10 was further decreased in senescent tumor cells. The gene discussed is CD8A; the disease is cancer.