Inflammatory cells infiltrate the sites of infection at an early stage of the infection with SARS-CoV2 and cause a stormy release of pro-inflammatory cytokines like IL-6, IL-17A, TNFα, IFNγ, IL-1α/β, and chemokines like CC-chemokine ligand 2 (CCL2) as well as CXC-chemokine ligand 10 (CXCL10) (13). This evidence concerns the gene IL1A and infection.