Our findings reiterate a previous study by Jones et al., which showed lean women primarily had augmented LHCGR expression and obese women overexpressed insulin receptor in adipose tissue corroborating that the pathophysiology of hyperandrogenemia in PCOS is different in lean and obese women and further confirming that obese women are more insulin resistant [8]. Here, LHCGR is linked to polycystic ovary syndrome.