In terms of its own function in cancer, it has been found that myosin-18A expression was increased in prostate cancer cells (Makowska et al., 2015), and its knockdown reorganized long NM2A-rich stress fibers and affected the cells’ ability to migrate (Makowska et al., 2015), suggesting a role of myosin-18A in prostate cancer metastasis (Peckham, 2016). This evidence concerns the gene MYO18A and prostate carcinoma.