Moreover, the majority of these PCATs were found to be also regulated by the AR, and analysis of previously published ChIP data (Pomerantz et al., 2015) revealed that most of the sites bound by ERG in PCATs were co-occupied by the AR (Kohvakka et al., 2020), confirming the previous findings by Yu and colleagues who reported the co-occupancy of AR and ERG in prostate cancer cells (Yu et al., 2010). The gene discussed is AR; the disease is Familial prostate cancer.