This study reveals that NR4A1 binds and recruits a corepressor molecule to the promoter regions of CD36 (also known as FA translocase) and fatty acid-binding protein 4 (FABP4), leading to transcriptional suppression, which hampers fatty acid uptake, leading to the inhibition of cell proliferation and impeding tumor cell growth in vivo (Yang et al., 2020). The gene discussed is NR4A1; the disease is neoplasm.