We found that hyperglycemia-induced OXTR reporter suppression occurred among the −2000, −1600, −1400, −1200, −1100, −800, −400 and −200 deletion constructs (numbered according to Ensembl gene ID: OXTR-201 ENST00000316793.7; transcription start site was marked as 0), while suppression was significantly restored in the −1000, and −900 deletion reporter constructs, indicating that hyperglycemia-responsive transcriptional element is located in the range of −1100∼−900 on the OXTR promoter (see Figure 2A). This evidence concerns the gene OXTR and Hyperglycemia.