The overexpression or hyperactivation of AKT (encoded by AKT1), epidermal growth factor receptor (EGFR; encoded by EGFR), or c-Jun (encoded by JUN) promotes various cancerous processes, including proliferation, growth, survival, invasion, and migration of BC cells and is further related to the poorer clinical outcomes of BC patients [102–127]. This evidence concerns the gene JUN and breast cancer.