Kaempferol, luteolin, and quercetin stimulate apoptotic cell death but inhibit cell processes, including proliferation, cell cycle progression, angiogenesis, migration, invasion, metastasis, and cancer stemness; such effects occur via the regulation of important BC-associated pathways such as the Akt, caspase, EGFR, estrogen, HER2, MAPK, insulin-like growth factor (IGF)-1, Notch, and Wnt signaling pathways [216–266]. The gene discussed is AKT1; the disease is breast cancer.