A recent study reported that C3C12 PPD, a newly discovered ginsenoside, inhibited cell growth, proliferation, migration, and tube formation of NSCLC in vivo and in vitro by inhibiting several signaling pathways, such as Raf/MEK/ERK, AKT/mTOR, and AKT/GSK-3β/β-catenin [113]. The gene discussed is MTOR; the disease is non-small cell lung carcinoma.