Expression of functional agrII in resistant pvl+ MSSA-CC5 and pvl+ MRSA-CC7 clones in pus, wound and abscess would further intensify invasiveness through action of enterotoxin genes (particularly sea, sec, sed, and sej), leukocidins (LukF-PV, lukD/lukE) and proteases (aur, slpA sspB, sspE, sspP). This evidence concerns the gene FUT2 and abscess.