For example, a study has recently shown that mice recapitulating the D252H missense mutation in neuronal translation-elongation factor eEF1A2, which is associated with neurodevelopmental disorders and late childhood epilepsy, do not show face validity for spontaneous seizures or neurodevelopmental deficits, despite the fact that human and murine eEF1A2 are almost identical (Davies et al., 2020). Here, EEF1A2 is linked to epilepsy.