It has been shown that cardiac muscle of HF individuals is subject to a gene regulatory shift, resulting in overexpression of 3‐hydroxybutyrate dehydrogenase 1 and , succinyl‐CoA 3‐oxoacid CoA transferase; enzymes which enhance the ketone oxidation pathway, as well as monocarboxylate transporter 1(Slc16a1), which increases the ketone transport capacity.4, 11. Here, SLC16A1 is linked to hydrops fetalis.