PDLIM7 and lupus nephritis: This suggests the possibility that enhanced and dysregulated cellular immunity associated with LMP1 functional mimicry (62, 67, 69, 71) may foster anti-Sm and anti-dsDNA autoantibody specificities associated with EBNA-1 molecular mimicry (35, 42–44) to propel some aspects of immune complex-driven lupus nephritis.