Previous findings have also shown a predominance of CCR5, CCR3, CCR2, and CCR1 in early infections, while expression of CXCR1 and CXCR2 occurred in late infections possibly released by activated monocytes and other inflammatory cells (Baggiolini, 2001; Borish and Steinke, 2003). The gene discussed is CCR2; the disease is infection.