Amyloid transthyretin (ATTR) amyloidosis is a progressively debilitating, clinically heterogeneous, and fatal disease caused by the buildup of transthyretin (TTR) amyloid fibrils in various organs and tissues, resulting in multisystem dysfunction particularly in the heart, along with the peripheral and autonomic nervous systems [1–3]. This evidence concerns the gene TTR and amyloidosis.