Dong et al. (2013) reported that elevation in Snail-SUV39H1 complex was in coordination with the elevation in H3K9me3 at the E-cadherin promoter leading to the recruitment of DNMT1 and gene silencing causing enhanced epithelial mesenchymal transition, a function of DNMT1 which is entirely different from the previously known biological function, in basal like breast cancer cell lines (Dong et al. 2013). This evidence concerns the gene SNAI1 and breast carcinoma.