Dong et al. (2013) reported that elevation in Snail-SUV39H1 complex was in coordination with the elevation in H3K9me3 at the E-cadherin promoter leading to the recruitment of DNMT1 and gene silencing causing enhanced epithelial mesenchymal transition, a function of DNMT1 which is entirely different from the previously known biological function, in basal like breast cancer cell lines (Dong et al. 2013). The gene discussed is SNAI1; the disease is breast cancer.