Moreover, TGF-β overexpression was found after brain death in the non-alcoholic steatotic liver, confirming previous reports showing that the grade of NAFLD activity, including hepatic steatosis, inflammation and fibrosis, is correlated with TGF-β expression level5,6, and HSCs can mediate the progression of NAFLD through TGF-β32–34. This evidence concerns the gene TGFB1 and Hepatic steatosis.