In addition, it is well documented that secondary IgAN is induced by various conditions, such as gastrointestinal disease, liver disorder, autoimmune disorders, and infection.[3] Among secondary IgAN, infection-related glomerulonephritis (IRGN) associated especially with staphylococcus species, induces glomerulonephritis due to the formation of IgA-dominant immunocomplex deposits. The gene discussed is CD79A; the disease is glomerulonephritis.