IgA nephropathy (IgAN) is the most common form of glomerulonephritis worldwide.[1] Galactose-deficient IgA1 (Gd-IgA1) is known to deposit specifically in the glomeruli of patients with IgAN.[2] However, histological features of IgAN is various as known in the Oxford Classification, which includes findings on light microscopy such as, mesangial hypercellularity, endocapillary hypercellularity, segmental glomerulosclerosis, tubular atrophy/interstitial fibrosis, and crescents. Here, IGHA1 is linked to IgA glomerulonephritis.