It is known that mucinous tumors, such as pancreatic, gastric, and ovarian tumors are capable of secreting enzymes that have abilities activating coagulation factor X. Remarkably, LMWH and rivaroxaban inhibited the coagulation factor Xa (FXa), hence preventing thrombosis, such that the microcirculation was improved, while the secondary consumption of platelets was alleviated.[4] Despite that LMWH exhibits an extremely low possibility of 0.06% to bring about heparin-induced thrombocytopenia (HIT), the decrease of platelets cannot be explained by HIT. Here, F10 is linked to mucinous neoplasm.