Although we did not observe any change in CD119 (IFN-γR) expression in Nr2f6-deficient OT-I cells (Fig. S6F), by d7 after infection, SLECs had accumulated at the expense of MPECs in the spleens of anti-IFN-γ treated mice, resulting in similar levels as compared to animals that had received isotype control (Fig. 6B). This evidence concerns the gene IFNG and infection.