At the myotube stage, a fibrosis‐related gene set was clearly up‐regulated in DMD cells, as illustrated by the overexpression of ANGPT1, CTGF, collagens (e.g. COL1A2), matrix metallopeptidases (MMPs), and tissue inhibitors of metallopeptidase (TIMPs) (Figure5B). The gene discussed is COL1A2; the disease is Duchenne muscular dystrophy.