CCN2 and Duchenne muscular dystrophy: Here, our extensive RNA‐seq data could help identify relevant therapeutic targets for pharmacological intervention, such as CTGF—involved in fibrosis and found up‐regulated in DMD myotubes—which can be inhibited by monoclonal antibodies,103 or TSPO receptor—a receptor potentially member of the mPTP down‐regulated in DMD cells—targetable with benzodiazepines.104