Former studies (Zhu et al., 2017) have reported that MEIS1 overexpression could induce non-apoptotic cell death of ccRCC cells via decreasing the levels of prosurvival regulators Survivin and BCL-2, and MEIS1 attenuates in vitro invasion and migration of ccRCC cells with downregulated epithelial–mesenchymal transition (EMT) process. The gene discussed is BCL2; the disease is nonpapillary renal cell carcinoma.