Coagulation dysfunction has been found in a high proportion of COVID-19 cases, as evidenced by increasing D-dimer levels and prolonged prothrombin time, as well as overt thrombotic manifestations (Guan et al., 2020; Helms et al., 2020; Huang C. et al., 2020; Tang et al., 2020; Wang D. et al., 2020). The gene discussed is F2; the disease is COVID-19.