Studies have found that the release of inflammatory cytokines, such as tumor necrosis factor (TNF-α) and interleukins (IL-1β and IL-6) increases, and promotes many immunopathological processes in sepsis, which are often referred to as “cytokines storm” (Ono et al., 2018; Hwang et al., 2020). This evidence concerns the gene IL6 and Sepsis.