Alterations in later ERP components have previously been detected in individuals with anxiety, Post-traumatic Stress Disorder (Johnson et al., 2013), ADHD (Szuromi et al., 2011), depressive disorders (Bruder et al., 2002), and autism (Cui et al., 2017), while alterations in the N100 component have been observed in individuals with schizophrenia (del Re et al., 2015) as well as in individuals with single-nucleotide polymorphism (SNPs) in another nicotinic acetylcholine receptor encoding gene, CHRNA4 (Mobascher et al., 2016). The gene discussed is CHRNA4; the disease is autism.